BafangFan t1_j3i4zu0 wrote
Reply to comment by calumin in New study challenges previous ideas regarding Alzheimer’s disease. Research found the increase in soluble Aβ may be a general aging-related change in the brain not specific to Alzheimer’s, while higher levels of fibrillary amyloid appear to be a better indicator of poorer brain health. by Wagamaga
Yeah, I/we could be wrong.
But the risks of diet change are very low. Maybe so diarrhea and cravings in the early stages of transition. Maybe some boredom of a limited diet.
But the rewards can be significant. People have gone from living in Groundhogs Day to being able to remember the past few months.
calumin t1_j3i6qjq wrote
Ok but this post about the science related to something completely different.
BafangFan t1_j3ie4jf wrote
I thought it was about the science of how to detect the degree of Alzheimer's in a person ?
I'm arguing that glucose metabolism is a more insightful indicator of Alzheimer's progression than any plaques/proteins.
And I'm arguing that if glucose metabolism is functioning poorly, the brain can alternatively use more fat/ketones; and with a non-impaired fuel source metabolism, the brain can restore some of its previous function.
calumin t1_j3imrll wrote
That’s not what the scientific article is about.
BafangFan t1_j3j4jkx wrote
>Rather than Alzheimer’s simply involving increased production of Aβ protein, the more important issue may be a reduced ability to effectively clear the protein and stave off the creation of plaque-contributing fibrillary amyloid, Thorwald said.
>“These findings further support the use of aggregated, or fibrillary, amyloid as a biomarker for Alzheimer’s treatments,” Thorwald said. “The site in which amyloid processing occurs has less precursor and enzyme available for processing, which may suggest the removal of amyloid as a key issue during Alzheimer’s.”
The focus is still on amyloid plaques, is it not?
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